The COVID Vaccine Can Make You Very Sick, And Now We Know Why
Spike protein
Introducing a vaccine against COVID-19, which literally makes our own cells produce the antigen (something that triggers an immune response, in this case, the spike protein), already sounded like a terrible idea. Now more and more data pours out, which confirms that this was really a bad idea.
How Do The Current COVID Vaccines Work?
I will describe it briefly, if you want to read more about this, you can read it here. The vaccine introduces an mRNA sequence in your body cells. Because of this, your own cells start to produce a spike protein and express this spike protein outside your own cells, but spike proteins also end up in your circulation. These spike proteins induce an immune reaction, and your body starts an immune response against this spike protein. This is how our immune system gets immunized against COVID. This technique is entirely new and bypasses all natural systems we have in our immune system. Originally this technique was developed as a vaccine against Ebola, which makes sense because we cannot treat Ebola, and Ebola has an infection mortality rate of 50% to 90%. But now, because of this man-made COVID crisis, we have introduced it also for an infection with an infection fatality rate of 0.24%.
Trouble In Paradise, How Smart Is It to Let Our Own Body Produce Spike Proteins?
There is medication that can reduce the severe symptoms of COVID. Still, the world governments have almost univocally decided that the only way out of this man-made crisis is vaccination. Governments don´t shy away to take measures that would fit a dictatorship, but we all have to bend to this, and universal rights are trampled en masse. Everything is aimed to get everybody vaccinated, and governments are not even shy to start vaccination of younger people and are already looking forward to having vaccinations once or twice a year. All this time, we still don´t know the long-term effect of this kind of vaccination (or gene therapy). The first problems started with healthy people who got thrombo-embolic complications after vaccination. The worrying part about this is that one of the thrombo-embolic processes is a cerebral venous sinus thrombosis, this is a very rare kind of thrombosis but appeared to appear more often after people got the COVID vaccine, 4 –14 days after vaccination. (Kowarz, Krutzke, & Reis, 2021) Another worrying thing is that even after vaccination, people still could develop a clinical picture which was like COVID and SARS (Severe Acute Respiratory Syndrome), and till now, we did not have a satisfying explanation for this.
Spike Protein, Quo Vadis?
So our cells start to produce spike proteins, and then what? The spike proteins are meant to give an immune response. The problem is that because our own cells are producing this, a lot of the usual steps in our immune response are bypassed, and the reaction we have is not a natural one. The spike proteins end up in our circulation, and that is a problem.
Spike Protein Damages Our Vascular Wall
The spike protein hinders the function of our endothelium (this is the inner layer in our blood vessels). The vascular endothelium is a key tissue that get infected and damaged by SARS-CoV-2. The function of the endothelium is hindered by diminishing the activity of ACE-2 (this is a very important enzyme, one of its functions is lowering blood pressure). The underlying mechanism involves spike protein downregulation causing ACE-2 destabilization. Thus, the spike protein-exerted vascular endothelial damage via ACE-2 downregulation overrides the decreased virus infectivity. It seems paradoxical that ACE-2 reduction by spike protein would decrease the virus infectivity, thereby protecting endothelium. However, a dysregulated renin-angiotensin system due to ACE-2 reduction may exacerbate endothelial dysfunction, leading to endotheliitis (an inflammation of the vascular wall). (Lei, Zhang, Schiavon , & et al., 2021) One of the other worrying things is that the spike protein alone is enough to cause disease. In a study, tissue samples showed inflammation in endothelial cells lining the pulmonary artery walls. (Lei, Zhang, Schiavon , & et al., 2021) The damage to the vascular wall could be an explanation for the thrombo-embolic complications. The interaction with the ACE-2 system should not have been a surprise because the SARS-CoV viruses use ACE-2 as an entry point into cells.
Spike Protein Damages Our Lungs
Using a newly developed mouse model of acute lung injury, researchers found that exposure to the spike protein alone was enough to induce COVID-19-like symptoms. This even included the severe inflammation of the lungs called SARS (Severe Acute Respiratory Syndrome). SARS-CoV2 spike protein causes lung injury even without the presence of the intact virus. The researchers found that the genetically modified mice injected with the spike protein exhibited COVID-19-like symptoms that included severe inflammation, an influx of white blood cells into their lungs, and evidence of a cytokine storm (an immune response in which the body starts to attack its own cells and tissues rather than just fighting off the virus). (Solopov, 2021)
Blood Cloths Cause Infarction; In Brain And In Abdomen
Severe side effects became overt during these vaccination campaigns: cerebral venous sinus thromboses (CVST), absolutely rare under normal conditions, were found as a severe side effect that occurred 4–14 days after the first vaccination. Besides CVST, Splanchnic Vein Thrombosis (SVT) was also observed; this is thrombosis of the veins in your abdomen. These events were mostly associated with thrombocytopenia and similar to the well-known Heparin-induced thrombocytopenia (HIT). Meanwhile, scientists have proposed a mechanism to explain this vaccine-induced thrombocytopenia. However, they do not provide a satisfactory explanation for the late thromboembolic events. The vaccine-induced thrombosis and thrombocytopenia (VITT) mechanism is not provoked by antibodies directed against the spike antigen. The proposed VITT mechanism explained the thrombocytopenia, but it did not explain the thromboembolic events.
The soluble spike variants initiate severe side effects when binding to ACE-2-expressing endothelial cells in blood vessels. In analogy to the thromboembolic events caused by spike protein encoded by the SARS-CoV-2 virus, the underlying disease mechanism is called the “Vaccine-Induced Covid-19 Mimicry” syndrome (VIC19M syndrome). Most of these cases display thrombocytopenia combined with a rare cerebral venous sinus thromboembolic event that caused the death in one third of affected vaccinated persons in Germany.
Our cells produce spike protein variants that have lost the important membrane anchor, resulting in secreted soluble spike protein variants which end up in our blood circulation. Soluble spike protein has been described to cause adverse effects, e.g., a strong inflammatory response on endothelial cells. Moreover, nearly all severe cases of SARS-CoV-2 infections (COVID-19) suffer from life-threatening thromboembolic events due to the many spike surface protein in the bloodstream. Even pseudoviruses with spike protein on the surface cause strong inflammatory reactions in tissues and endothelial cells, indicating the danger of this protein. When this spike protein ends up in our circulation, such thromboses may occur in any site of the human body where endothelial cells express ACE-2. When the immune system starts to produce antibodies against the spike protein, the endothelial cells will not only bind the soluble spike protein variants but would also be attacked with the newly formed antibodies. This will give rise to strong inflammatory reactions. (Kowarz, Krutzke, & Reis, 2021)
Why We Should Not Give It to Younger People
If this mechanism is correct, we can also understand why elderly people experience less complications. Elderly people may frequently use drugs that decrease blood coagulation and inflammatory events (aspirin or vitamin K inhibitors such as Marcumar). This could be a reason why elderly people suffer much less from these thromboembolic events. In addition, an older immune system displays more immune aging, which means that young people exhibit stronger immune reactions than elderly people, and women even stronger than men. All this would imply a higher incidence in young women when compared to men or elderly people. (Kowarz, Krutzke, & Reis, 2021)
This Experiment Has To Stop
There is mounting evidence that the new type of mRNA vaccine can have serious side effects and may even cause death. There is now more and more evidence that the problem is the spike protein, which is exactly what our own cells produce after vaccination. The vaccine is not the only treatment for COVID-19, and there are alternatives. It is a crime to make this vaccine directly or indirectly mandatory, and it defies all medico-ethical rules we have in our society. This vaccine is still in phase 3, and we don´t know the long-term effect. No one should be forced to be part of a medical experiment.
Interesting Links:
Before You Take the COVID-19 Vaccine, Read This, It Will Blow Your Mind!
The COVID-19 Numbers No One Talks About
COVID-19, It Is Not the Virus That Is Killing Us
Why Mandatory Vaccination Is a Very Bad Idea
References
Kowarz, E., Krutzke, L., & Reis, J. (2021). “Vaccine-Induced Covid-19 Mimicry” Syndrome: Splice reactions within the SARS-CoV-2 Spike open reading frame result in Spike protein variants that may cause thromboembolic events in patients immunized with vector-based vaccines. Research Square. doi:https://doi.org/10.21203/rs.3.rs-558954/v1
Lei, Y., Zhang, J., Schiavon , C., et al. (2021, 3 31). SARS-CoV-2 Spike Protein Impairs Endothelial Function via Downregulation of ACE2. bioRxiv, 128(9), 1323–1326. doi:https://doi.org/10.1161/CIRCRESAHA.121.318902
Solopov, P. (2021). R4183 — Single intratracheal exposure to SARS-CoV-2 S1 spike protein induces acute lung injury in K18-hACE2 transgenic mice. Experimental Biology 2021.
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